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Dead fawn this morning help answer my question

if it was lung worm (i have never heard of it so i could sound dumb) what causes it and can it pass from deer to deer
 
Ivermectin Plus, SafeGuard, and Valbazen all treat lungworm.

(DO NOT give Valbazen wormer to pregnant does during the first 45 days of pregnancy~I don't give Valbazen at all during any stage of pregnancy)

I Prefer Ivermectin Plus Injectable If I think they have a problem.

I use SafeGuard Flakes (expensive at first but lasts long $300. a bucket)as prevention.



Remember Wormer is Poison You Give enough poison to kill the parasite but (hopefully) not enough to kill the host......Be especially careful when worming sick animals, because of this.

Also

If the deer is REALLY infested you can kill them by killing worms to quickly.





Robert if that works for you I AM NOT SAYING CHANGE IT....

I am just stating facts so people can chose what MAY work best for them...

I prefer to use Sulmet and Bovatec (Very Limited)on my farm...I have Corid To mix it up every once in a while...But Corid Should not be used every day as it ALSO becomes ineffective.

Corid is a Coccidia killer..This is the only medication that will actually kill Coccidia...Problem with it is it also wipes out the Thaimine in the body in order to be effective.

To prevent that... People would give B Complex with the Corid, which was like shooting themselves in the foot, because for Corid to work, it has to wipe out the Thaimine that has Coccidia attached to it...Corid was designed and marketed for cattle, which seem to handle a TEMPORARY lack of Thaimine better than other livestock.



I MYSELF FEEL THIS IS ONE OF THE LARGEST KILLER OF DEER/FAWNS

Here is info on Goat Polio!!

http://www.jackmauldin.com/health/goat_polio.htm





The official name for Goat Polio is "Polioencephalomalacia". It is a nutritional /metabolic disease that affects primarily ruminants, including goats. Metabolic means relating to metabolism, the whole range of biochemical processes that occur within the goat's rumen. In ruminant animals that means processing the food the goat has eaten. This disease has become more prominent in goats in the U.S. that are under intensive management conditions when they are fed more concentrated feed to get them to grow faster.



The main cause of this disease is either a thiamine deficiency or the stopping of the thiamine activity in the rumen. Thiamine, or vitamin B1, is a major part of the rumen processing that occurs with carbohydrates and amino acid in the rumen. Decreased processing activity leads to cell death in the brain and swelling from excessive accumulation of watery fluid in brain cells and tissues. The symptoms originate from the damage to the brain.



The thiamine is produced in sufficient amounts by specific bacteria in the goat's rumen. Any changes in the normal environment in the rumen may lead to reduced production of thiamine in the rumen Normally something else has occurred to upset the goat's processing of its food intake and that causes the Goat Polio to occur.



Causes



Things that can cause the rumen to not produce the required thiamine are:

Rumen acidosis cause by excessive concentrate feeding and sudden feed changes.

TOO MUCH CORN can cause Acidosis Especially during warm weather



Moldy feeds.



Thiabendazole - active ingredient in some wormer medication like Thibenzole Sheep & Goat Wormer



Levamisole - wormer



Some types of ferns



Following a shot of an antibiotic. - the antibiotic is used for killing bad bacteria but may also kill the good bacteria in the rumen that helps process the feed that results in thiamine. ALWAYS FOLLOW UP ANY ANTIBIOTIC SHOT WITH A TREATMENT OF "PROBIOS" some similar product that will repopulate the rumen with good microbes.



Overdosing of Amprolium - ingredant in medication such as CORID for prevention of coccidiosis: Amprlium is a thiamine analog, competitively inhibits the active transport of thiamine.



Feeding horse type of sweet feed with high molasses content.



Signs of Goat Polio



Depression



Not eating and/or diarrhea



Early neurologic dysfunction such as



excitability



elevation of the head



staring off into space



aimless wandering



circling



muscle tremors



apparent blindness



As the disease progresses



Involuntary eye movement



extensor rigidity - the muscles that extends or straightens a limb or body part



convulsions



Diagnosis



Early signs of depression and diarrhea are also common in enterotoxemia and pregnancy toxemia. If the animal is not close to kidding, you can rule out the pregnancy toxemia. The blindness and/or extensor rigidity are strong symptoms for goat polio instead of enterotoxemia. The extensor rigidity is also a sign of tetanus. If the animal is circling, that is also a sign of listeriosis. The best thing to consider is what lead up to the symptoms and quickly watching for any signs of blindness. Look at the list of what can cause the goat polio and consider if any of them have occurred recently. Also, it will not hurt to treat an animal with tetnus or listeriosis with the treatment for goat polio.
 
Bj is right again! to much typing and forget the smallest things sometimes make several trips and stil dont rember DC
 
BillieJo,

I agree with your post. However, the treatment with B1 (Thiaimine) is not without risk. Anaphalacticn (Sp?) shock can occur in priomarily young deer and goats. An Epinephrin injection is a MUST as a standby to counteract shock.
 
i was talking to my vet about putting wormer in their water at all times and he said that if your not careful about this the worm itself can develop a immunity to the medicine. by giving them small doses that might not necessarily kill them. it only makes it worse.
 
Scott.... I agree But....if you look at past Posts, I said I never give B injections on young deer.... I give oral Paste YOU CAN EASILY KILL A FAWN WITH VITAMIN B...But above I was saying You hurt yourself if you give Vit~B because that is how corid works.
 

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