Tell me things are not changing with CWD!

Yea and terry is on boone and crockets facebook as del feldon Trying to act like he is part of them dividing hunters

Just look at the barnyard fertilizer ignorant chart that the QDMA has released about EHD and CWD. Science does not support the chart. I assume that the info was provided to the QDMA by John R Fischer. The QDMA has taken a position against deer farming. Deer farms are being supported by every hunter that purchases deer urine. The chart claims that CWD is spread by deer urine and feces. Scientific research has proven that this is false. People are beginning to see that CWD is being used as a scam.

http://health.ucsd.edu/news/releases/Pages/2015-02-23-human-prion-protein-and-chronic-wasting-disease.aspx


 


Small Loop in Human Prion Protein Prevents Chronic Wasting Disease

Finding provides new therapeutic target for prion diseases


February 24, 2015  |  Heather Buschman, PhD 


Chronic wasting disease (CWD) — an infectious disease caused by prions — affects North American elk and deer, but has not been observed in humans. Using a mouse model that expresses an altered form of the normal human prion protein, researchers at University of California, San Diego School of Medicine have determined why the human proteins aren’t corrupted when exposed to the elk prions. Their study, published Feb. 23 in the Journal of Clinical Investigation, identifies a small loop in the human prion protein that confers resistance to chronic wasting disease.

brown prion proteins

Prion protein aggregates (brown) in the brain of a mouse expressing the human-elk protein.

“Since the loop has been found to be a key segment in prion protein aggregation, this site could be targeted for the development of new therapeutics designed to block prion conversion,� said Christina Sigurdson, DVM, PhD, associate professor at UC San Diego and UC Davis and senior author of the study.

Prions aren’t microorganisms like bacteria or viruses; they’re simply protein aggregates. Some prion diseases are caused by an inherited genetic mutation, while others are caused by exposure to infectious prions in food. Acquired prion diseases are triggered when a foreign, misfolded prion protein causes the body’s own natural prion proteins to misfold and aggregate. In addition to chronic wasting disease, examples include scrapie and bovine spongiform encephalopathy (or "mad cow disease") in animals and variant Creutzfeldt-Jakob disease in humans. In humans, prion diseases can cause a variety of rapidly progressive neurological symptoms, such as difficulty walking and speaking, and dementia. These diseases are 100 percent fatal and there is currently no effective treatment.

“We suspected that a loop in the human prion protein structure may block the elk prions from binding, as the sequences did not appear to be compatible,� Sigurdson said.

To test this hypothesis, Sigurdson and her team developed a transgenic mouse that expresses a prion protein that’s identical to the human version — except for a small loop, which they swapped out for the elk prion sequence. When these mice were exposed to the elk prions, they developed chronic wasting disease.

In contrast, control mice expressing the normal human prion sequence resisted infection when exposed to same materials — just as humans seem to, even those who consume venison meat.

“This finding suggests that the loop structure is crucial to prion conversion and that sequence compatibility with the host prion protein at this site is required for the transmission of certain prion diseases,� Sigurdson said.

Co-authors of this study include Timothy D. Kurt, Cyrus Bett, Jun Liu, Tom Yang, UC San Diego; Lin Jiang, David Eisenberg, UC Los Angeles and Howard Hughes Medical Institute; Natalia Fernández-Borges, CIC bioGUNE, Spain; Terry R. Spraker, Colorado State University, Fort Collins; Joaquín Castilla, CIC bioGUNE and Basque Foundation for Science, Spain; and Qingzhong Kong, Case Western Reserve University.

This research was funded, in part, by the National Institutes of Health (grants NS055116, NS069566, U54AI0359 and AG029430), national grants from Spain and the Morris Animal Foundation.

If 99% deer don't get sickly or die like they thought they would and humans don't get it what are we doing to ourselves with the CWD program???  


the program was asked for by the deer industry 


CWD is not a reportable disease

The state may pay for the CWD test but we pay our vet for every sample she takes. In the case of the Brakke's, when their deer were tested there was a high incident of positives, but none of the deer were sick. Can a deer be a carrier and never get sick?

How do we know when we kill them all?

Wisc hunters say maybe 5% in the highly positive zones showed any signs a of sickness