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Not so good news !!!!!!

Jarrid, it's interesting that some of the first cases of CWD where found near the Research facilities near Fort Collins, Co. Also Wisconsin, look where the big outbreak was, near the research farm at the U of W Madison. Coincidences ????

Gary
 
Another thing here in Wi , was the DNR pushed and pushed the issue of how horrible CWD would be to our deer herd because they wanted the Fed's to give/grant them 25 million for their own lab. The fed's didn't buy into that 25 million idea and told the DNR the state had a good enough lab at the UW Madison campus so they didn't get one but they pushed the all deadly idea so they could get one . Just hasn't worked for them so instead of them testing they're just making us do it for them at our exspense !
 
PRWhitetails said:
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3199251/



Could the spontaneity of cwd throughout the country be related in someways to the presence of scrapie or areas that had sheep herds that were identified to have scrapie?? I found this paper, and what I get out of it is that strains of scrapie can be contracted by deer, and that their is little difference in the symptoms when compared to "cwd". Could deer possibly contract strains of scrapie and because it is found in deer, it is called cwd? I found in another paper that there were over 900 herds of sheep throughout the country that had scrapie in their animals (paper was from the 90's..ill try to find it again), but cannot find anything that shows where these herds were/are. Also, does anyone know of any other research done on more natural modes of transmisson of scrapie to deer that have been done? I dont mean to ramble but I think this just continues to show the many possible modes of "movement" of cwd.



I doubt anyone could deny the strong link between Scrapie & the origin of CWD.



Sharkey
 
Heres what they say so far!



Response to new cases of Pennsylvania deer with chronic wasting disease pending







By Marcus Schneck | [email protected]

on March 04, 2013 at 4:15 PM, updated March 04, 2013 at 4:25 PM

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State agencies provided a bit more detail about three new Pennsylvania cases of deer with chronic wasting disease, but little in the way of the actions they will take in response to those cases, at a press conference Monday afternoon.



View full sizeA total of 2,089 samples were collected from hunter-killed deer in the disease management area designated last fall by the Pennsylvania Game Commission across portions of Adams and York counties.DAN GLEITER, The Patriot-News



The three new confirmed cases of CWD – all in free-ranging deer – were detected in an adult buck in Frankstown Township, Blair County; an adult doe in Freedom Township, Blair County; and a year-and-a-half-old buck in South Woodbury Township, Bedford County, according to Brad Meyers, southcentral regional director of the Pennsylvania Game Commission.



The hunters who harvested those deer did not notice anything usual about them, he said, noting, "Our officers asked them if these deer appeared sick or were acting strangely, and there was no evidence of that."



CWD is an always fatal brain disease among white-tailed deer and other cervids, like elk and moose.



Although it does not recommend that anyone eat venison from a deer confirmed to be infected with CWD, the Centers for Disease Control says, there is no evidence that the disease can be spread to other types of animals or to humans.



According to Calvin DuBrock, director of the commission's Bureau of Wildlife Management, all test results on samples taken from hunter-killed deer in the most recent hunting seasons in that area of southcentral Pennsylvania have been received, and only those three came back as confirmed for CWD.



In addition, results have come back as negative on all 2,089 deer sampled from within the 400-square-mile disease management area in Adams and York counties that the commission established last fall after Pennsylvania's first case of CWD was confirmed in a captive herd of deer.



Priority was placed on samples from the DMA and then samples from the area of Bedford and Blair counties because of their proximity to a spreading area of CWD in Maryland, about 10 miles south of the Mason-Dixon line with Bedford County.



Other results, from more than a thousand additional samples taken from hunter-killed and road-killed deer farther north in the state, have not yet been received, said DuBrock.



"We're expecting results over the next several weeks," he explained.



That timeframe also applies to the schedule the commission, the Pennsylvania Department of Agriculture and other partners on the state's CWD Response Task Force expect to follow in setting the state's response to the three new cases of CWD.



"We're going to increase the testing of free-ranging deer in that area" through additional sampling of road-killed deer, deer killed for crop damage and recovery of deer reported as appearing sick, said DuBrock.



However, the full, official response that will be detailed in an executive order by the commission has not yet been determined.



"We're going to have to lay that out in the coming months," he said. "It will be spelled out in the executive order in the days ahead. There needs to be a lot more discussion within our agencies and within the task force."



"At the extreme end is what we did in Adams County this past year," plus anything else the task force determines to be necessary, he said.



However, he cautioned, "can we continue to spend $300,000-500,000 in each disease management area? It was pretty expensive."



The impact on deer farms within the three new areas of concern also remains up in the air, according to Craig Schultz, director of the Bureau of Animal Health within the department of agriculture.



"We've already started work on determining the coordinates for all facilities" in the affected area, and will be working with the Game Commission to determine the best course of additional action, he said.



"Once we have those results (from tests on all samples collected from hunter-deer killed), I think we'll have a better idea moving forward," he explained.



DuBrock said a public meeting, similar to meetings held last fall in Adams and York counties, has been tentatively scheduled for somewhere in Bedford or Blair County.



And a little more!



PGC press conference today identified the 3 free-ranging CWD positive deer as an adult buck, an adult doe and a yearling buck. The adult buck and doe were found in Blair Co., the yearling buck in Bedford Co.



Closest distance to known existing free ranging CWD positive deer in Maryland, approx. 20 miles, approx. 50 miles from CWD positive captive deer in Pennsylvania.



CWD task force will be deciding what regulatory changes will be implemented as part of the management plan after the remaining samples are tested, although they don't anticipate any further positives being found.
 
The impact on deer farms within the three new areas of concern also remains up in the air, according to Craig Schultz, director of the Bureau of Animal Health within the department of agriculture

Wow, that's really comforting! I wouldn't be surprised if they decide to depopulate all the deer farms in these counties, whose to say they won't consider it? Even more good news for me, one of the positives came from within my township. I am in Freedom Township.

The article says they want to test as many deer as possible in these counties, I imagine my pen of captive deer will be a great starting point for them. Sorry, I may be overreacting, but I must say, I am a little scared about this.
 
Also I was not aware anyone in my township was submitting samples on harvested deer, I was not told I had to, so I assume it must be a voluntary thing. One thing is for sure, my dream of having fun raising and selling deer is about over, there will be little to no farmers that are going to want to buy from a guy with a farm in the same township as wild deer with CWD. I try to keep a positive attitude about things, but man, what a complete slap in the face of bad luck for me lately. First I buy from the Adams county farm, my doe gets shot for testing (no compensation for me) then I am quarantined for a couple months, sales had to be cancelled on some of my bred doe, now wild deer have CWD, not only in my State, not only in my County, but in my EXACT township!! Give me a break!!
 
Antlershed.....you know my thoughts jand I will continue to pray for you and your farm....there are some BIG Time deer farmers in your same situation and I will not name anyone....however, I will just say......keep a close eye on them......if you are approached and they (the Dept. Of Ag.) try to put the hammer down on you....it needs to be the same for everyone within your area.....BIG....SMALL.....or what ever size they are.....there are some BIG BOYS that have a lot of pull in our State and there is one that is close to you that will be in the same boat....so I guess what I am trying to say is.....if they want to try to okay hard they better be doing it with everyone....BiG.....SMALL or whom ever......I will do anything I can to help.....my number is 412-287-5534......just call me for anything and I will see what I can do.......praying for you Brother!
 
Matt and I are in the same exact boat. And in their article they talk of an executive order going to be brought down in a few days. I think we all know what that means. I have already had people driving down on my private property to my deer pens and leaving before I can make it out of the house what is next? This by far is not the dream I had when I started raising deer for my son. This is heartbreaking and to make matters worse I am out of town all week to try and comfort my family who is just waiting for the order to "depopulate" I mean slay in cold blood
 
This is a reminder of what we went through back in 2001 with CWD and testing. Deer farming has never had a brighter future from the hunting side of things. We are finally getting the snowball building and getting hunters to see preserves are a quality experience. The CWD is a major set back and a big bullet coming at us. If we sit on our hands im sure we will get hit hard. Everything we have accomplished has been with an aggressive offense. Could the deer farms affected by this be the sleeping giant?



I believe I will be deer farming for many years to come. I hope everyone on here feels the same. I would be greatful to provide our lodge and food for us to get together and get on the same page.
 
I am also willing to open up my restauraunt to hold any meetings that anyone would like to put together, we can seat up to 150 in a private room. We are located about 10 min outside of Harrisburg. Just give me a call.

717-805-3799

Thank you,
 
champs01 said:
I am also willing to open up my restauraunt to hold any meetings that anyone would like to put together, we can seat up to 150 in a private room. We are located about 10 min outside of Harrisburg. Just give me a call.

717-805-3799

Thank you,



That would never hurt, Thats for sure. These cases were in the wild and 20 miles from CWD in Maryland. We have to remember that and push that point. CWD could have walked into Pa!
 
ddwhitetails said:
Antlershed.....you know my thoughts jand I will continue to pray for you and your farm....there are some BIG Time deer farmers in your same situation and I will not name anyone....however, I will just say......keep a close eye on them......if you are approached and they (the Dept. Of Ag.) try to put the hammer down on you....it needs to be the same for everyone within your area.....BIG....SMALL.....or what ever size they are.....there are some BIG BOYS that have a lot of pull in our State and there is one that is close to you that will be in the same boat....so I guess what I am trying to say is.....if they want to try to okay hard they better be doing it with everyone....BiG.....SMALL or whom ever......I will do anything I can to help.....my number is 412-287-5534......just call me for anything and I will see what I can do.......praying for you Brother!



Dennis,

What does that mean some of the Big Boys have pull in our state?
 
First of all Rodger my post was not meant in any hateful or jealous manor.....so maybe you need to do some soul searching within yourself if you thought it was.........I was simply saying that there are some within our State that have a lot more pull with the Dept. Of Ag. Than others.......I know this as a fact as I have heard it straight from the horses mouth.......I am not jealous of this fact it simply is what it is.....my advice to Antlershed was simply to use that to his advantage if need be......nothing more and nothing less......I certianly do not know what the big deal is about saying....Big Deer Farmer or Small Deer Farmer........The fact is there are Bigger Deer Farmers and Smaller Deer Farmers I don't see it as any kind of divide as others do(without mentioning names!) just as in many other Industries there are Big Cattle Farmers and Small Cattle farmers....big pig farmers and small pig farmers.....big chicken farmers and small chicken farmers...what's the big deal seriously......there are enough issues goin on within the Industry lately and I am honestly just trying to help give Antlershed a little hope in a very bad situation that he has been dealt........I hope I answered your question Josh.....there was no malicious intent with my post.....
 
There is a lot of evidence that points to you being wrong. Prion diseases are most likely a result of a virus. Example:



Yale School of Medicine professor Dr. Laura Manuelidis, the head of neuropathology at the school, and her team of researchers recently published a report in the Proceedings of the National Academy of Sciences asserting that a virus, rather than prion proteins, is the cause of mad cow disease in animals and Creutzfeldt-Jakob disease in humans. These spongiform encephalopathies of the brain have traditionally been thought to be caused by prions * abnormal proteins that convert healthy proteins to the disease state. But the new study suggests prions may simply be part of the late stages of the diseases, not part of the causes.



Transmissible spongiform encephalopathies affect the brains and nervous systems of victims, including sheep, deer and humans. The term ”spongiform” comes from the fact that the infection causes neurons to die, leaving tiny holes in the brain so that it eventually resembles a sponge. Humans afflicted with the incurable degenerative disease show memory and personality changes and sometimes problems with movement. Mad cow, the cattle version of the disease, has achieved infamy because the infectious agent in cows appears to be the cause of vCJD, a variant form of CJD in humans.



The research team’s goal was to try to identify viral particles in infected cells. They infected cell lines with either scrapie (a sheep disease related to mad cow) or CJD agents and found virus-like particles that did not contain prion protein. An abundance of these particles was related to high levels of infectivity, which was not true of the presence of prion proteins.



“People hypothesize that prion proteins are infectious, but they’re probably part of the disease, not the infectious agent itself,” Manuelidis said.



The virus-like particles had been found by other researchers but were largely ignored. They were first identified in 1968 in synaptic regions of scrapie-infected brain and later found in many other animals with different TSEs. But Manuelidis said that researchers apparently forgot about them once the prion hypothesis became dominant.



“I had totally forgotten about them, too,” she said. “But after we found the 25-nanometer particles, I went home and remembered I’ve seen these before. I went back to the old journals from the ’70s and there they were.”



Manuelidis said part of the reason for the neglect of the virus-like particles is that in previous studies, they were always found in degenerating brain tissue, so it was impossible for scientists to draw accurate conclusions about them. What allowed her to collect relevant experimental data was the fact that her team observed the particles in highly infectious tissue culture cells.



“We saw them in tissue culture cells that weren’t degenerating,” she said. “The infected cells were as healthy as the uninfected cells that had no 25-nm virus-like particles.”



The study is a work in progress, and the researchers want to conduct more experiments with tissue cultures to gather further evidence and learn more about the particles. Manuelidis said that since it is easier to work with a simplified cell system than with infected animals whose brains are degenerating, tissue culture experiments can be used to identify essential features of the infectious agent and clarify the way in which the particles invade cells. Her team will use the tissue cultures to purify the virus-like particles more completely.



“In the future, we will try to isolate the particles from tissue cultures and characterize what is in them,” she said.



The team’s specific viral particle hypothesis suggests new avenues for treatment and vaccines, Manuelidis said. If they are successful in rapidly measuring infectivity in tissue culture, they will be able to gain better understanding of possible remedies. She even pointed out that prion proteins, while not the infectious agent, are probably essential receptors for replication and growth of the TSE virus.



”The infectious agent needs prion proteins to grow,” Manuelidis said. “This means targeting the prion protein may also be a useful therapeutic step.”



Researchers at other universities pointed out that the Yale study does not definitely prove the viral hypothesis, nor does it fully disprove the prion hypothesis.



Tricia Serio, an assistant professor of molecular biology, cell biology and biochemistry at Brown University, said that while the research is intriguing, the viral hypothesis still needs to be directly proven. She also pointed out that there are many examples of protein-based phenotypes, like the one described in the prion hypothesis.



“For example, we study prions in yeast, and work from the Weissman, King, Liebman, Saupe and Wickner labs has shown that the transfer of recombinant protein produced in bacteria is sufficient to induce a heritable phenotype in yeast,” she said. ”This is direct proof of a prion mechanism for the yeast traits.”



Surachai Supattapone, associate professor of biochemistry and medicine at Dartmouth University, said that the next challenge for Dr. Manuelidis’ group will be to isolate and identify a specific virus that can cause transmissible spongiform encephalopathy. He said, however, that there is still work ahead for prion protein researchers.



“Proponents of the ‘protein only’ hypothesis * which is also not proven * will need to demonstrate that purified prion proteins alone can fulfill Koch’s postulates [a set of criteria for establishing a causal relationship between an infectious agent and a disease] to prove their alternative claim,” he said.



http://www.global-sisterhood-network.org/content/view/1565/59/



sharkey said:
Thanks Kurt. Good info.



For anyone who is going to get involved in lobbying for abatement actions, CWD should not be compared to EHD. I'll say it again, raising a negative point to try to sell a positive one, only raises the negative.



CWD is a prion disease, not a virus. CWD is not close to reaching its critical mass potential, nor is it clear if it will jump species in the way other prion diseases have. CWD does not just spontainiously appear,nor is it "everywhere". There is going to be many routes of infection & playing one stakeholder off againt another,just gives this disease more time. Stay informed.



Sharkey
 

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